e-ISSN: 2321-3647
logo

American Journal of Pharmacy and Health Research

Published

Role of Viruddha Ahara in the Genesis of Amavata

Published in March 2015 Issue 3 (Vol. 3, Issue 3, 2015)

Role of Viruddha Ahara in the Genesis of Amavata - Issue cover

Abstract

Those substances that cause an unnatural increase in the humors but do not expel them out of body are called Viruddha ahara. Hence, they are more antagonistic than nourishing to the tissues. Many of food materials mentioned in this context in treatises cannot be identified. The interaction is more important. There is concept of drug-drug or food-drug interaction in modern medicine also. In today’s era most of these incompatibilities can be explained chemically and ultimately, leading to ama formation in body. Amavata develops due to conglomeration of ama and vata. According to Ayurveda, ama plays an important role in the pathogenesis of this disease, which is formed mainly due to decrease in digestive power. Acharya Madhava has described various types of nidana responsible for the development of Amavata and viruddha ahara is one of them which plays the most crucial point in today’s era. Viruddha ahara causes vitiation in agni causing vitiation in doshas and vice-versa.

Authors (3)

Saini Neera

Ph.D Scholar, Dept of Vikriti ...

View all publications →

Pal Pradeep Kumar

Junior Redident, Dept of Vikri...

View all publications →

Byadgi P S

Asst. Professor, Dept of Vikri...

View all publications →

Download Article

PDF

Best for printing and citation

File size: 0.0 MB
Format: PDF

Download Article

PDF

Best for printing and citation

File size: 0.0 MB
Format: PDF

Article Information

AJPHR303001

AJPHR-30-000001

2015-03-01

Article Impact

Views:4,832
Downloads:2,287

How to Cite

APA
MLA
Chicago
Harvard
Vancouver
IEEE
ACM
ACS
ABNT
Turabian
RIS (EndNote/Zotero/Mendeley)
BibTeX

Neera & Pradeep, P. & P, B. (2015). Role of Viruddha Ahara in the Genesis of Amavata. American Journal of Pharmacy and Health Research, 3(3), xx-xx. https://ajphr.com/articles/AJPHR303001

Article Actions